a(CHEBI:"calcium(2+)", loc(GO:intracellular)) regulates bp(GO:"signal release")

PubMed:19126755

It was then recognized that Ca2+ flux directly through nAChR channels or indirectly via voltage-gated Ca2+ channels is relevant for nicotinic modulation of transmitter release, synaptic plasticity, as well as neuronal viability, differentiation, and migration.

Related Edges 3

• a(CHEBI:"calcium(2+)", loc(GO:intracellular)) regulates bp(MESH:"Neuronal Plasticity")
• a(CHEBI:"calcium(2+)", loc(GO:intracellular)) regulates bp(GO:"neuron migration")
• a(CHEBI:"calcium(2+)", loc(GO:intracellular)) regulates bp(GO:"neuron differentiation")

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