TNFRSF

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Entity

Name: TNFRSF
Namespace: FPLX
Namespace Version: 20180917
Namespace URL: https://raw.githubusercontent.com/sorgerlab/famplex/e8ae9926ff95266032cb74f77973c84939bffbeb/export/famplex.belns

Out-Edges 1

p(FPLX:TNFRSF) increases p(HGNC:NLRP3) View Subject | View Object

Many innate immune signalling or cytokine receptors, such as the TNFR, activate transcription of NLRP3 and thereby influence the susceptibility of immune cells to NLRP3 inflammasome triggers37,38 (FIG. 2). PubMed:23702978

Appears in Networks:

Activation and regulation of the inflammasomes v1.0.0

Annotations

Confidence: High
NeuroMMSigDB: Tumor necrosis factor subgraph

About

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.

`p(FPLX:TNFRSF)`