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p(HBP:"AK1 R132A")

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Entity

Name
AK1 R132A
Namespace
HBP
Namespace Version
None
Pattern
.*

Appears in Networks 1

Neuropathogenic role of adenylate kinase-1 in Aβ-mediated tau phosphorylation via AMPK and GSK3β. v1.0.0

In-Edges 0

Out-Edges 1

p(HBP:"AK1 R132A") causesNoChange p(ECCODE:"2.7.11.31", pmod(Ph, Thr, 172)) View Subject | View Object

AK1 overexpression markedly decreased AMPK phosphorylation at Thr172 in the primary neurons, whereas AK1 R132A mutant failed to do so PubMed:22419736

Appears in Networks:

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BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.