p(MGI:Dock8) positiveCorrelation p(MGI:Cdc42)

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PubMed:27798618

In contrast with wild-type ER-HoxB8 BMDMs, Dock8−/− ER-HoxB8 BMDM did not spread or extend filopodia (Fig. 7a,b), and failed to activate Cdc42 following treatment with heme (Fig. 7c), indicating that DOCK8 was essential for heme-induced activation of Cdc42.

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• p(MGI:Cdc42) positiveCorrelation p(MGI:Dock8)

Cell Ontology (CL)

monocyte

MeSH

Blood

MeSH

Sepsis

Text Location

Results

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