1. Catalog
  2. Nodes
  3. p(HGNC:VPS35, var("?"))

p(HGNC:VPS35, var("?"))

Equivalencies: 0 | Classes: 0 | Children: 0 | Explore

Entity

Name
VPS35
Namespace
hgnc
Namespace Version
20180906
Namespace URL
https://raw.githubusercontent.com/pharmacome/terminology/b46b65c3da259b6e86026514dfececab7c22a11b/external/hgnc-names.belns

Appears in Networks 1

Alzheimer’s disease and the autophagic-lysosomal system v1.0.0

In-Edges 3

p(HGNC:VPS35) hasVariant p(HGNC:VPS35, var("?")) View Subject | View Object

Appears in Networks:

path(MESH:"Alzheimer Disease") association p(HGNC:VPS35, var("?")) View Subject | View Object

VPS35 mutations have been shown to disrupt macroautophagy [113] and mitochondrial function [114] and are associated with AD and PD [102,115]. PubMed:29758300

Appears in Networks:

path(MESH:"Parkinson Disease") association p(HGNC:VPS35, var("?")) View Subject | View Object

VPS35 mutations have been shown to disrupt macroautophagy [113] and mitochondrial function [114] and are associated with AD and PD [102,115]. PubMed:29758300

Appears in Networks:

Out-Edges 4

p(HGNC:VPS35, var("?")) decreases bp(GO:macroautophagy) View Subject | View Object

VPS35 mutations have been shown to disrupt macroautophagy [113] and mitochondrial function [114] and are associated with AD and PD [102,115]. PubMed:29758300

Appears in Networks:

p(HGNC:VPS35, var("?")) decreases act(a(GO:mitochondrion)) View Subject | View Object

VPS35 mutations have been shown to disrupt macroautophagy [113] and mitochondrial function [114] and are associated with AD and PD [102,115]. PubMed:29758300

Appears in Networks:

p(HGNC:VPS35, var("?")) association path(MESH:"Alzheimer Disease") View Subject | View Object

VPS35 mutations have been shown to disrupt macroautophagy [113] and mitochondrial function [114] and are associated with AD and PD [102,115]. PubMed:29758300

Appears in Networks:

p(HGNC:VPS35, var("?")) association path(MESH:"Parkinson Disease") View Subject | View Object

VPS35 mutations have been shown to disrupt macroautophagy [113] and mitochondrial function [114] and are associated with AD and PD [102,115]. PubMed:29758300

Appears in Networks:

About

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.