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bp(MESH:"Energy Metabolism")

Equivalencies: 0 | Classes: 0 | Children: 0 | Explore

Entity

Name
Energy Metabolism
Namespace
mesh
Namespace Version
20180828
Namespace URL
https://raw.githubusercontent.com/pharmacome/terminology/1b20f0637c395f8aa89c2e2e342d7b704062c242/external/mesh-names.belns

Appears in Networks 2

Nuclear receptors as therapeutic targets for Alzheimer's disease. v1.0.0

The Biology of Proteostasis in Aging and Disease v1.0.0

In-Edges 5

p(FPLX:PPAR) association bp(MESH:"Energy Metabolism") View Subject | View Object

PPARs have been shown to play essential roles in energy metabolism, adipocyte differentiation, insulin sensitization and tumor suppression. PubMed:21718217

Appears in Networks:
Annotations
Confidence
High
MeSH
Microglia

p(FPLX:PPAR) positiveCorrelation bp(MESH:"Energy Metabolism") View Subject | View Object

PPARs have been shown to play essential roles in energy metabolism, adipocyte differentiation, insulin sensitization and tumor suppression. PubMed:21718217

Appears in Networks:
Annotations
Confidence
Medium

p(HGNC:PPARG) association bp(MESH:"Energy Metabolism") View Subject | View Object

PPAR-g is involved in lipid storage, insulin sensitivity and energy metabolism and has been shown to promote adipocyte differentiation [21]. PubMed:21718217

Appears in Networks:
Annotations
Confidence
High
MeSH
Microglia

p(HGNC:PPARGC1A) association bp(MESH:"Energy Metabolism") View Subject | View Object

PPAR-g is involved in lipid storage, insulin sensitivity and energy metabolism and has been shown to promote adipocyte differentiation [21]. PubMed:21718217

Appears in Networks:
Annotations
Confidence
Medium

p(HGNC:HSPD1) association bp(MESH:"Energy Metabolism") View Subject | View Object

HSP60 is essential for maturation and maintenance of the mitochondrial proteome and is therefore intimately linked to energy production. PubMed:25784053

Appears in Networks:
Annotations
MeSH
Mitochondria

Out-Edges 5

bp(MESH:"Energy Metabolism") association p(FPLX:PPAR) View Subject | View Object

PPARs have been shown to play essential roles in energy metabolism, adipocyte differentiation, insulin sensitization and tumor suppression. PubMed:21718217

Appears in Networks:
Annotations
Confidence
High
MeSH
Microglia

bp(MESH:"Energy Metabolism") positiveCorrelation p(FPLX:PPAR) View Subject | View Object

PPARs have been shown to play essential roles in energy metabolism, adipocyte differentiation, insulin sensitization and tumor suppression. PubMed:21718217

Appears in Networks:
Annotations
Confidence
Medium

bp(MESH:"Energy Metabolism") association p(HGNC:PPARG) View Subject | View Object

PPAR-g is involved in lipid storage, insulin sensitivity and energy metabolism and has been shown to promote adipocyte differentiation [21]. PubMed:21718217

Appears in Networks:
Annotations
Confidence
High
MeSH
Microglia

bp(MESH:"Energy Metabolism") association p(HGNC:PPARGC1A) View Subject | View Object

PPAR-g is involved in lipid storage, insulin sensitivity and energy metabolism and has been shown to promote adipocyte differentiation [21]. PubMed:21718217

Appears in Networks:
Annotations
Confidence
Medium

bp(MESH:"Energy Metabolism") association p(HGNC:HSPD1) View Subject | View Object

HSP60 is essential for maturation and maintenance of the mitochondrial proteome and is therefore intimately linked to energy production. PubMed:25784053

Appears in Networks:
Annotations
MeSH
Mitochondria

About

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.