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p(MGI:Dapk1)

Equivalencies: 0 | Classes: 0 | Children: 0 | Explore

Entity

Name
Dapk1
Namespace
mgi
Namespace Version
20181021
Namespace URL
https://raw.githubusercontent.com/pharmacome/terminology/f2f993e599694ab5ce989cc39d789a499f75db99/external/mgi-names.belns

Appears in Networks 1

Tau Modifications v1.9.5

Tau Modifications Sections of NESTOR

In-Edges 2

p(MGI:Mapt) association p(MGI:Dapk1) View Subject | View Object

Direct interaction of DAPK1 with Tau causes spine loss and subsequently neuronal death. DAPK1 phosphorylates Tau protein at Ser262 (pS(262)) in cortical neurons of stroke mice. PubMed:25995053

Appears in Networks:
Annotations
MeSH
Stroke

complex(p(MGI:Dapk1), p(MGI:Mapt)) hasComponent p(MGI:Dapk1) View Subject | View Object

Appears in Networks:

Out-Edges 3

p(MGI:Dapk1) association p(MGI:Mapt) View Subject | View Object

Direct interaction of DAPK1 with Tau causes spine loss and subsequently neuronal death. DAPK1 phosphorylates Tau protein at Ser262 (pS(262)) in cortical neurons of stroke mice. PubMed:25995053

Appears in Networks:
Annotations
MeSH
Stroke

p(MGI:Dapk1) directlyIncreases complex(p(MGI:Dapk1), p(MGI:Mapt)) View Subject | View Object

Direct interaction of DAPK1 with Tau causes spine loss and subsequently neuronal death. DAPK1 phosphorylates Tau protein at Ser262 (pS(262)) in cortical neurons of stroke mice. PubMed:25995053

Appears in Networks:
Annotations
MeSH
Stroke

p(MGI:Dapk1) increases p(MGI:Mapt, pmod(Ph, Ser, 262)) View Subject | View Object

Direct interaction of DAPK1 with Tau causes spine loss and subsequently neuronal death. DAPK1 phosphorylates Tau protein at Ser262 (pS(262)) in cortical neurons of stroke mice. PubMed:25995053

Appears in Networks:
Annotations
Uberon
cerebral cortex
MeSH
Stroke

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If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.