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bp(GO:"L-glutamate import")

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Entity

Name
L-glutamate import
Namespace
go
Namespace Version
20190207
Namespace URL
https://raw.githubusercontent.com/pharmacome/terminology/a5b84013a08880975ca84f40999e4404b14a97e2/external/go-names.belns

Appears in Networks 2

The Ubiquitin–Proteasome System and the Autophagic–Lysosomal System in Alzheimer Disease v1.0.0

The autism/neuroprotection-linked ADNP/NAP regulate the excitatory glutamatergic synapse v1.0.0

In-Edges 2

a(HBP:AβOs) decreases bp(GO:"L-glutamate import") View Subject | View Object

They also facilitate long-term depression by, among other effects, disrupting synaptic glutamate uptake (Li et al. 2009). PubMed:22908190

Appears in Networks:
Annotations
MeSH
Synapses

p(HGNC:SLC17A7) increases bp(GO:"L-glutamate import") View Subject | View Object

VGLUT1 is both necessary and sufficient for uptake and storage of glutamate, and thus comprises the sole determinant for an excitatory glutamatergic phenotype PubMed:30664622

Appears in Networks:

Out-Edges 0

About

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.