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  4. Extracellular Fluid

Extracellular Fluid

Name
Extracellular Fluid
Namespace Keyword
MeSHAnatomy
Namespace
MeSH
Namespace Version
20170511
Namespace URL
https://arty.scai.fraunhofer.de/artifactory/bel/annotation/mesh-anatomy/mesh-anatomy-20170511.belanno

Sample Annotated Edges 5

bp(HP:Neurodegeneration) decreases sec(p(HGNC:MAPT)) View Subject | View Object

Recently, tau was discovered in the interstitial fluid of awake, wild-type mice, suggesting its release by neurons in the absence of neurodegeneration (Yamada et al., 2011) PubMed:28420982

Appears in Networks:
Annotations
Confidence
Medium
MeSH
Extracellular Fluid

a(HP:"glymphatic system") decreases p(HGNC:MAPT) View Subject | View Object

It has been reported that ISF tau can be eliminated by the glymphatic system and the function of this clearance mechanism may be impaired due to the loss of AQP4 after TBI, which ultimately accelerates tau accumulation (Iliff et al.2014) PubMed:29626319

Appears in Networks:
Annotations
MeSH
Extracellular Fluid

path(MESH:"Brain Injuries, Traumatic") decreases a(HP:"glymphatic system") View Subject | View Object

It has been reported that ISF tau can be eliminated by the glymphatic system and the function of this clearance mechanism may be impaired due to the loss of AQP4 after TBI, which ultimately accelerates tau accumulation (Iliff et al.2014) PubMed:29626319

Appears in Networks:
Annotations
MeSH
Extracellular Fluid

path(MESH:"Brain Injuries, Traumatic") decreases p(HGNC:AQP4) View Subject | View Object

It has been reported that ISF tau can be eliminated by the glymphatic system and the function of this clearance mechanism may be impaired due to the loss of AQP4 after TBI, which ultimately accelerates tau accumulation (Iliff et al.2014) PubMed:29626319

Appears in Networks:
Annotations
MeSH
Extracellular Fluid

p(HGNC:AQP4) increases a(HP:"glymphatic system") View Subject | View Object

It has been reported that ISF tau can be eliminated by the glymphatic system and the function of this clearance mechanism may be impaired due to the loss of AQP4 after TBI, which ultimately accelerates tau accumulation (Iliff et al.2014) PubMed:29626319

Appears in Networks:
Annotations
MeSH
Extracellular Fluid

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About

BEL Commons is developed and maintained in an academic capacity by Charles Tapley Hoyt and Daniel Domingo-Fernández at the Fraunhofer SCAI Department of Bioinformatics with support from the IMI project, AETIONOMY. It is built on top of PyBEL, an open source project. Please feel free to contact us here to give us feedback or report any issues. Also, see our Publishing Notes and Data Protection information.

If you find BEL Commons useful in your work, please consider citing: Hoyt, C. T., Domingo-Fernández, D., & Hofmann-Apitius, M. (2018). BEL Commons: an environment for exploration and analysis of networks encoded in Biological Expression Language. Database, 2018(3), 1–11.